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As for low-density lipoproteins (LDL), N-homocysteination produces aggregation, thus the accumulation of cholesterol, and facilitates the mediated absorption of oxidized LDL by macrophage scavenger receptors, resulting in the formation of foam cells in atherosclerosis [43–44]

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This may cause cloudiness, visible particles, clumping, foaming, gel formation, aggregation, or precipitation

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