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In conclusion, we explored a potential mechanism that the delayed healing of diabetic wounds was closely related to the downregulation of circCDK13, and circCDK13 promoted the proliferation and migration of HDFs and HEKs through forming circCDK13-IGF2BP3-mRNA ternary complex, which could stabilize mRNA and upregulate CD44 and c-MYC (Fig
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These soluble signals are secreted by various stromal components, including CAFs, immune cells, and endothelial cells, and act on CSCs
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