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To identify upstream kinases responsible for this activation, we focused on MST1—previously implicated in FAS-mediated STK38 phosphorylation [23]—and MEKK2, which has been reported to interact with STK38 [25, 26] and has also been shown to activate the STK38-related kinase LATS in an RIPK1-dependent manner following TNF-α stimulation [45]

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WGBS profiling reveals that hypomethylation of the OsZIP1 promoter specifically excludes Cd, Cu and Zn from shoots, while maintaining essential nutrient levels