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PRIMA-1 has been reported to inhibit carcinogenesis and promote the expression of target genes such as Puma, Bax, and CDKN1A

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Santa Maria et al., 2014), thus supporting the idea that a fully mutated FMR1 transcript can have toxic effects and that elongated transcripts are causative for FXTAS, but not for FXS, although some mRNA is present in many FXS patients (Tassone et al., 2001)