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Driven by persistent hyperglycemia, target organ damage in T2D results from cellular stress, overproduction of reactive oxygen species (ROS), advanced glycation end products (AGEs), and protein kinase C (PKC) activation, ultimately leading to the development of microvascular and macrovascular diseases
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Size:300 ct. (60 servings)
receives funding from the European Research Council (ERC-CoG Trusted no.101044445), the German Research Foundation (DFG TRR296, TRR152, SFB1123 and GRK 2816/1) and the German Center for Diabetes Research (DZD e.V.)