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The administration of vitamin E before and after an OVA sensitization and challenge decreases not only oxidative stress and its consequences, including the development of airway inflammation (eosinophilia and the levels of pro-inflammatory cytokine levels such as IL-4, 5, and 13), but also AHR in OVA mice [384,403,415], especially in the presence of oxidative stress enhancers such as oxidized graphene, where vitamin E administration decreases Th2 responses [395]