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Moreover, the cell surface of alkaline phosphatase activity, which is a marker enzyme of oxidant-producing intracellular compartments (OPICs), is also suppressed by gliotoxin in human neutrophils stimulated with phorbol myristate acetate (PMA) as well as the repression of NADPH oxidase [61], thus contributing to the inhibition of superoxide production in PMA-treated human neutrophils
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