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Guided by the coordinated upregulation of glucose transporters such as GLUT1 and rate-limiting enzymes including hexokinase II (HK2), phosphofructokinase-1 (PFK-1), pyruvate kinase M2 (PKM2) and lactate dehydrogenase A (LDHA), these tumours support anabolic diversion of glycolytic intermediates while simultaneously depleting the local glucose pool 47 (Table 1)
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