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examined the contribution of estrogens in maintaining cognition in women, which has also been reported by other investigators, and provided convincing evidence implicating the menopause-related decline in 17β-estradiol in cognitive aging and AD risk [173]
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Figure 4 Alterations in the intracellular redox status are often associated with GSH depletion (Polonikov, 2020) and endogenous GSH deficiency, due either to decreased biosynthesis and/or increased consumption, is a marked contributor to the pathogenesis of numerous diseases via mechanisms including oxidative stress and inflammation
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