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synthesized a negative peptide ERAP (11R-GGG-QMLSDLTLQLRQR) which could release PHB2 from BIG3 and peptide ERAP treatment enhanced tamoxifen responsiveness in ERa-positive breast cancer cells (Yoshimaru et al., 2013)

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While these data provide valuable biological plausibility, many proposed pathways—particularly those involving mitochondrial function, substrate partitioning, and micronutrient dynamics—remain insufficiently validated in well-controlled human studies