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A landmark clinical trial published in 2010 demonstrated that H₂-enriched dialysis fluid reduced oxidative stress markers in patients undergoing hemodialysis.7 Subsequent research explored various administration methods—such as inhalation, H₂-rich drinking water, H₂-generating nanomaterials, and H₂-releasing solid-state compounds—across diverse experimental and clinical settings.8,9 Additionally, investigations have consistently confirmed that H₂ selectively targets harmful radicals, reduces inflammation, and beneficially modulates cellular signaling pathways implicated in cardiovascular, neurodegenerative, and renal disorders.10,11 Recent studies have observed the beneficial role of H₂ in renal disorders through mechanisms involving redox modulation and inflammatory response regulation.8,9,12,13,14,15 A recent review observed H₂ as a potential intervention in CKD, highlighting its modulation of oxidative stress-related pathways including: (i) the nuclear factor erythroid 2-related factor 2–Kelch-like ECH-associated protein 1 complex (Nrf2–Keap1)

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