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Furthermore, acetylcholine-induced production of bioavailable nitric oxide was lower in aortic rings from GPx1 −/− ApoE mice than ApoE −/− mice, and the double knockout mice had increased 3-nitrotyrosine staining in atheromatous plaques, similar to earlier findings of endothelial dysfunction in GPx1-deficient mice [39, 70, 71]
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Furthermore, evidence in the rats has so far shown the recovery of the various muscle disabilities deriving from a multitude of different causes, peripheral and central, given that muscle weakness was part of the primary disturbance that was also attenuated
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